Involvement of lipid microdomains in human endothelial cells infected by Streptococcus agalactiae type III belonging to the hypervirulent ST-17.
Beatriz Jandre FerreiraPamella Silva Lannes-CostaGabriela da Silva SantosCláudia MermelsteinMarcelo Einicker-LamasPrescilla Emy NagaoPublished in: Memorias do Instituto Oswaldo Cruz (2020)
The collection of our results suggests that lipid microdomain affects the interaction of S. agalactiae type III belonging to the hypervirulent ST-17 with HUVEC cells through PI3K/Akt signaling pathway.
Keyphrases
- type iii
- pi k akt
- cell cycle arrest
- endothelial cells
- signaling pathway
- induced apoptosis
- klebsiella pneumoniae
- cell proliferation
- high glucose
- fatty acid
- epithelial mesenchymal transition
- cell death
- multidrug resistant
- vascular endothelial growth factor
- biofilm formation
- pluripotent stem cells
- endoplasmic reticulum stress
- escherichia coli
- staphylococcus aureus