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AKAP12 Upregulation Associates With PDE8A to Accelerate Cardiac Dysfunction.

Hanan QasimMehrdad RajaeiYing XuArfaxad Reyes-AlcarazHala Y AbdelnasserM David StewartSatadru K LahiriXander H T WehrensBradley K McConnell
Published in: Circulation research (2024)
AKAP12 upregulation in cardiac tissue is associated with accelerated cardiac dysfunction through the AKAP12-PDE8 axis.
Keyphrases
  • left ventricular
  • poor prognosis
  • cell proliferation
  • oxidative stress
  • signaling pathway
  • atrial fibrillation