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Osteoarthritic infrapatellar fat pad aggravates cartilage degradation via activation of p38MAPK and ERK1/2 pathways.

Zuoqing ZhouSu'an TangXiaoyu NieYiqun ZhangDelong LiYang ZhaoYumei CaoJianwen YinTianyu ChenGuangfeng RuanZhaohua ZhuXiaochun BaiWeiyu HanChanghai Ding
Published in: Inflammation research : official journal of the European Histamine Research Society ... [et al.] (2021)
Osteoarthritic IPFP induces the degradation and inflammation of cartilage via activation of p38MAPK and ERK1/2 pathways, in which IL-1β and TNF-α act as the key factors. Our study suggests that modulating the effects of IPFP on cartilage may be a promising strategy for knee OA intervention.
Keyphrases
  • signaling pathway
  • extracellular matrix
  • cell proliferation
  • pi k akt
  • knee osteoarthritis
  • randomized controlled trial
  • oxidative stress
  • total knee arthroplasty
  • adipose tissue
  • fatty acid