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In vivo metabolomics identifies CD38 as an emergent vulnerability in LKB1 -mutant lung cancer.

Jiehui DengDavid H PengDavid FenyoHao YuanAlfonso LopezDaniel S LevinMary MeynardieMari QuinterosMichela RanieriSoumyadip SahuSally C M LauElaine ShumVamsidhar VelchetiSalman R PunekarNatasha RekhtmanCatríona M DowlingVajira K WeerasekaraYun XueHongbin JiYik SiuDrew JonesAaron N HataTakeshi ShimamuraJohn T PoirierCharles M RudinTakamitsu HattoriShohei KoideThales PapagiannakopoulosBenjamin G NeelNabeel BardeesyKwok-Kin Wong
Published in: bioRxiv : the preprint server for biology (2023)
tumor suppressor of lung adenocarcinoma patients and are associated with resistance to current treatments. Our study identified CD38 as a potential therapeutic target that is highly overexpressed in this specific subtype of cancer, associated with a shift in NAD homeostasis.
Keyphrases
  • end stage renal disease
  • newly diagnosed
  • ejection fraction
  • chronic kidney disease
  • prognostic factors
  • mass spectrometry
  • nk cells
  • peritoneal dialysis
  • gene expression
  • genome wide
  • young adults