Majid Hafezparast

Scopus Author ID ORCID

Publication Activity (10 Years)

Years Active: 2019-2024
Publications (10 Years): 11

Top Venues

Disease models & mechanisms

Journal of neuroinflammation

Annals of medicine

Amyotrophic lateral sclerosis & frontotemporal degeneration

University of Sussex

University of Sussex (PHD)

This page only lists publications with an associated author ORCID identifier.

Affiliation History

University of Sussex Apr 01 2019

United Kingdom

Professor of Molecular Neuroscience :: School of Life Sciences

University of Sussex Jul 01 2006 - Mar 31 2019

United Kingdom

Reader in Human/Mammalian Molecular Genetics :: School of Life Sciences

University of Sussex Aug 01 2003 - Jun 30 2006

United Kingdom

Lecturer in Human/Mammalian Molecular Genetics :: School of Life Sciences

University College London Oct 01 2001 - Jul 31 2003

United Kingdom

Senior Research Fellow :: Institute of Neurology

Imperial College London Dec 01 1997 - Sep 30 2001

United Kingdom

Postdoctoral Research Fellow :: Prion Disease Unit

University College London Oct 01 1992 - Nov 30 1997

United Kingdom

Postdoctoral Research Fellow

Education History

University of Sussex Oct 01 1990 - Sep 30 1993

United Kingdom

PHD :: Medical Research Council Cell Mutation Unit

King's College London Jan 10 1987 - Jun 30 1990

United Kingdom

BSC :: Biochemistry

Publications

Eleni Christoforidou, Libby Moody, Greig Joilin, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
An ALS-associated mutation dysregulates microglia-derived extracellular microRNAs in a sex-specific manner. Disease models & mechanisms 17 (5) (2024)
Eleni Christoforidou, Libby Moody, Greig Joilin, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
The ALS-associated TDP-43M337V mutation dysregulates microglia-derived extracellular microRNAs in a sex-specific manner. Disease models & mechanisms (2024)
Eleni Christoforidou, Libby Moody, Greig Joilin, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
An ALS-associated mutation dysregulates microglia-derived extracellular microRNAs in a sex-specific manner. Disease models & mechanisms 17 (5) (2024)
Eleni Christoforidou, Libby Moody, Greig Joilin, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
An ALS-associated mutation dysregulates microglia-derived extracellular microRNAs in a sex-specific manner. Disease models & mechanisms 17 (5) (2024)
Eleni Christoforidou, Libby Moody, Greig Joilin, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
An ALS-associated mutation dysregulates microglia-derived extracellular microRNAs in a sex-specific manner. Disease models & mechanisms 17 (5) (2024)
Eleni Christoforidou, Libby Moody, Greig Joilin, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
An ALS-associated mutation dysregulates microglia-derived extracellular microRNAs in a sex-specific manner. Disease models & mechanisms 17 (5) (2024)
Eleni Christoforidou, Fabio A Simoes, David Gordon, Kevin Talbot, Majid Hafezparast
Aberrant dynein function promotes TDP-43 aggregation and upregulation of p62 in male mice harboring transgenic human TDP-43. Amyotrophic lateral sclerosis & frontotemporal degeneration (2023)
Greig Joilin, Elizabeth Gray, Alexander Guy Thompson, Kevin Talbot, P Nigel Leigh, Sarah F Newbury, Matthew C Kiernan, Majid Hafezparast
Profiling non-coding RNA expression in cerebrospinal fluid of amyotrophic lateral sclerosis patients. Annals of medicine 54 (1) (2022)
Greig Joilin, Elizabeth Gray, Alexander Guy Thompson, Yoana Bobeva, Kevin Talbot, Jochen Weishaupt, Albert Ludolph, Andrea Malaspina, P Nigel Leigh, Sarah F Newbury, Martin R Turner, Majid Hafezparast
Identification of a potential non-coding RNA biomarker signature for amyotrophic lateral sclerosis. Brain communications 2 (1) (2020)
Eleni Christoforidou, Greig Joilin, Majid Hafezparast
Potential of activated microglia as a source of dysregulated extracellular microRNAs contributing to neurodegeneration in amyotrophic lateral sclerosis. Journal of neuroinflammation 17 (1) (2020)
Maria Isabel Martinez-Macias, Duncan Aq Moore, Ryan L Green, Fernando Gomez-Herreros, Marcel Naumann, Andreas Hermann, Philip Van Damme, Majid Hafezparast, Keith W Caldecott
FUS (fused in sarcoma) is a component of the cellular response to topoisomerase I-induced DNA breakage and transcriptional stress. Life science alliance 2 (2) (2019)

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Publication Activity (10 Years)

Top Topics

Top Venues

Majid Hafezparast