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MOZ and Menin-MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor.

Matthew L HemmingMorgan R BensonMichael A LoycanoJustin A AndersonJessica L AndersenMadeleine L TaddeiAndrei V KrivtsovBrandon J AubreyJevon A CutlerCharles HattonEwa SicinskaScott A Armstrong
Published in: Cancer discovery (2022)
Although many malignancies rely on oncogene amplification, GIST instead depends upon epigenetic regulation of KIT and other essential genes. Utilizing genome-scale CRISPR dependency screens, we identified complementary chromatin-modifying complexes essential to GIST and characterize the consequences of their disruption, elucidating a novel therapeutic approach to this disease. This article is highlighted in the In This Issue feature, p. 1599.
Keyphrases
  • genome wide
  • dna methylation
  • transcription factor
  • gene expression
  • acute myeloid leukemia
  • machine learning
  • deep learning
  • dna damage
  • nucleic acid
  • crispr cas
  • single cell
  • label free
  • genome editing