Merridee A Wouters

ResearcherID Scopus Author ID ORCID

Publication Activity (10 Years)

Years Active: 2015-2020
Publications (10 Years): 5

Top Topics

Molecular Dynamics Simulations

Mass Spectrometry

Binding Protein

Congenital Heart Disease

Top Venues

Nature communications

Protein science : a publication of the Protein Society

This page only lists publications with an associated author ORCID identifier.

Publications

Naomi L Haworth, Michael J Wouters, Morgan O Hunter, Lixia Ma, Merridee A Wouters
Cross-strand disulfides in the hydrogen bonding site of antiparallel β-sheet (aCSDhs): Forbidden disulfides that are highly strained, easily broken. Protein science : a publication of the Protein Society 28 (1) (2020)
Neil David Young, Tiffany J Harris, Marco Evangelista, Sharon Tran, Merridee A Wouters, Tatiana P Soares da Costa, Nadia J Kershaw, Robin B Gasser, Brian J Smith, Erinna F Lee, Walter Douglas Fairlie
Diversity in the intrinsic apoptosis pathway of nematodes. Communications biology 3 (1) (2020)
Rebecca C Poulos, Peter G Hains, Rohan Shah, Natasha Lucas, Dylan Xavier, Srikanth S Manda, Asim Anees, Jennifer M S Koh, Sadia Mahboob, Max Wittman, Steven G Williams, Erin K Sykes, Michael Hecker, Michael Dausmann, Merridee A Wouters, Keith Ashman, Jean Yee Hwa Yang, Peter J Wild, Anna De Fazio, Rosemary L Balleine, Brett J Tully, Ruedi Aebersold, Terence P Speed, Yansheng Liu, Roger R Reddel, Phillip J Robinson, Qing Zhong
Strategies to enable large-scale proteomics for reproducible research. Nature communications 11 (1) (2020)
Kaavya A Mohanasundaram, Mani P Grover, Tamsyn M Crowley, Andrzej Goscinski, Merridee A Wouters
Mapping genotype-phenotype associations of nsSNPs in coiled-coil oligomerization domains of the human proteome. Human mutation 38 (10) (2017)
Romaric Bouveret, Ashley J Waardenberg, Nicole Schonrock, Mirana Ramialison, Tram Doan, Danielle de Jong, Antoine Bondue, Gurpreet Kaur, Stephanie Mohamed, Hananeh Fonoudi, Chiann-Mun Chen, Merridee A Wouters, Shoumo Bhattacharya, Nicolas Plachta, Sally L Dunwoodie, Gavin Chapman, Cédric Blanpain, Richard P Harvey
NKX2-5 mutations causative for congenital heart disease retain functionality and are directed to hundreds of targets. eLife 4 (2015)