BRD-810 is a highly selective MCL1 inhibitor with optimized in vivo clearance and robust efficacy in solid and hematological tumor models.
Ulrike RauhGuo WeiMichael Serrano-WuGeorgios KosmidisStefan KaulfussFranziska SiegelKai ThedeJames McFarlandChristopher T LemkeNicolas WerbeckKatrin Nowak-ReppelSabine PilariStephan MenzMatthias OckerWeiqun ZhangKyle DavisGuillaume Poncet-MontangeJennifer A RothDouglas S DanielsVirendar K KaushikBrian HubbardKarl ZiegelbauerTodd R GolubPublished in: Nature cancer (2024)
The MCL1 gene is frequently amplified in cancer and codes for the antiapoptotic protein myeloid cell leukemia 1 (MCL1), which confers resistance to the current standard of care. Therefore, MCL1 is an attractive anticancer target. Here we describe BRD-810 as a potent and selective MCL1 inhibitor and its key design principle of rapid systemic clearance to potentially minimize area under the curve-driven toxicities associated with MCL1 inhibition. BRD-810 induced rapid cell killing within 4 h in vitro but, in the same 4-h window, had no impact on cell viability or troponin I release in human induced pluripotent stem cell-derived cardiomyocytes, even at suprapharmacologic concentrations. In vivo BRD-810 induced efficacy in xenograft hematological and solid tumor models despite the short residence time of BRD-810 in plasma. In totality, our data support the hypothesis that short-term inhibition of MCL1 with BRD-810 can induce apoptosis in tumor cells while maintaining an acceptable safety profile. We, therefore, intend to advance BRD-810 to clinical trials.
Keyphrases
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